However, when the regulation of the activity of the immune system is impaired or under chronic, long-term exposure to pathogenic microorganisms, the described processes go out of control, which leads to excessive proliferation (hypertrophy) of the lymphoid tissue. ([email protected]) It is worth noting that the protective properties of the hypertrophied tonsil are significantly reduced, as a result of which it itself can be colonized by pathogenic microorganisms, that is, become a source of chronic infection.

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As the child grows (especiallyespecially after 3 years, when children begin to attend kindergartens and be in crowded places) his immune system comes into contact with an increasing number of new microorganisms, which can lead to an overactive immune system and the development of adenoids.

In some children, palatine tonsil enlargement may be asymptomatic until adulthood, while in other cases, breathing problems and other symptoms of the disease may develop. Congenital developmental anomalies. In the process of Ivermectin formation in the intrauterine period, various disorders can be noted, which can be provoked by environmental factors (for example, polluted atmospheric air, high radiation levels), injuries or chronic diseases of the mother, abuse of alcoholic beverages or drugs (by the mother or father of the child).

This may result in a congenital enlargement of the nasopharyngeal tonsil. A genetic predisposition to adenoids is also not excluded, however, there is no specific data confirming this fact. Frequent infectious diseases. Chronic or often recurrent (stromectol) diseases of the upper respiratory tract (tonsillitis, pharyngitis, bronchitis) can lead to dysregulation of the inflammatory process in the lymphoid ring of the pharynx, which may result in an increase in the nasopharyngeal tonsil and the appearance of adenoids. A special risk in this regard is posed by acute respiratory viral diseases (ARVI), that is, colds, flu.

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Allergic diseases.
The mechanisms of inflammation in infection and in the development of allergic reactions are largely similar.

In addition, the immune system of an allergic child is initially predisposed to more pronounced reactions in response to the penetration of infection into the body, which can also contribute to hypertrophy of the pharyngeal tonsil.

Harmful environmental factors.
If a child breathes air contaminated with dust or harmful chemical compounds for a long time, this can lead to non-infectious inflammation of the lymphoid formations of the nasopharynx and the proliferation of adenoids.
Symptoms of adenoids are: impaired nasal breathing; runny nose; stromectol; hearing impairment; increased body temperature; deformation of the face.
Violation of nasal breathing with adenoids.
As the disease progresses, it becomes more difficult for the child to breathe, and his general condition worsens. Due to oxygen starvation and inadequate sleep, a pronounced lag in mental and physical development may appear.
More than half of children with adenoids have regular nasal discharge.
The reason for this is the excessive activity of the immune organs of the nasopharynx (in particular, the nasopharyngeal tonsil), as well as the constantly progressive inflammatory process in them.
This leads to an increase in the activity of the goblet cells of the nasal mucosa (these cells are responsible for the production of mucus), which causes the appearance of a cold.

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In this case, it becomes impossible to equalize the pressure between the tympanic cavity and the atmosphere. The air from the tympanic cavity is gradually absorbed, as a result of which the mobility of the tympanic membrane is impaired, which causes hearing loss.

Hearing impairment with adenoids.

Hearing impairment is associated with excessive growth of the nasopharyngeal tonsil, which in some cases can reach enormous size and literally block the internal (pharyngeal) openings of the auditory tubes.

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